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And this undisclosed SIV variant "could affect the results presented in this Research Article." .You must declare any conflicts of interest related to your comments and responses.Please see our Commenting Guide for further information.If the virus had repaired itself and the stop codon was gone, Ansari said, perhaps there's some mechanism, some dynamic between the monkeys' own immune systems and the anti-alpha4beta7 treatment that could still offer hope as an avenue of investigation. But I'm trying to point out the fact that [NIAID] got the virus from Villinger and got different results." "I'm reluctant to say any field is dead; alpha4beta7 is an important pathway in HIV infection," he said."But whether an antibody to alpha4beta7 can lead to consistent, long-term suppression of the virus — that is clearly now questionable." Comments on Medscape are moderated and should be professional in tone and on topic.The scope of the journal highlights the advanced topics in drug development, HIV vaccines, pre-exposure prophylaxis, or post-exposure prophylaxis, Post-exposure prophylaxis, Sex Education, HIV/AIDS Denialism, AIDS Dementia Complex, rural livelihoods, stigma, patient oriented research studies, and other significant new studies related to the field of HIV/AIDS & Clinical Research.Scientific Journal of HIV/AIDS & Clinical Research (SJHCR) publishes Original research articles, clinical trials, Reviews, Case report, Editorials, Letter to the editor, Short communication, Opinion, Book reviews and other special featured articles related to the field of HIV/AIDS & Clinical Research."If you have to place animals on [antiretroviral treatment] before 2 weeks because the virus is too virulent (ie, a study I am currently involved in), to ensure animals have an immune response worth salvaging therapeutically, the value of such a model is, in my mind, even more questionable," he told Yet "we did not have any animals that totally suppressed SIV in the absence of antiretroviral drug," he said.In a presentation of that study at the 2018 International AIDS Conference, the researchers make clear that they used the gene from all the monkeys to see whether the stop codon was still present in the monkeys before they began antiretroviral therapy and then the anti-alpha4beta7 treatment. "I'm the first to admit that something funny is going on.He's the one who provided the virus for both animal studies.The conflict, then, isn't over whether the mutated version of SIVmac239 was used and not specifically stated in the paper.